Voluntary activation level (VAL) is a neurophysiological measure quantifying the extent to which an individual can consciously recruit and activate their motor units during a maximal voluntary muscle contraction. This level is a direct index of the nervous system’s efficiency in driving the muscle to its full potential, distinct from the muscle’s intrinsic contractile strength. Clinically, a suboptimal VAL suggests a central nervous system limitation, rather than a peripheral muscle weakness, which is crucial for differential diagnosis and targeted rehabilitation strategies.
Origin
This term originates from neurophysiology and exercise science, specifically using techniques like the interpolated twitch technique to assess the integrity of the descending motor drive. The concept recognizes that maximum muscle force is often limited by neural factors, not just muscle mass or hormonal status. It is a critical metric for understanding the central component of physical performance and age-related strength decline.
Mechanism
The mechanism involves the precise integration of cortical motor commands and spinal motor neuron excitability, leading to the synchronized recruitment of the maximum possible number of motor units within a muscle. A low VAL indicates that the descending motor signal is insufficient to fully excite all motor neurons, leaving some muscle fibers unrecruited. Factors like central fatigue, pain, or poor neuroendocrine signaling can modulate the descending drive, thus reducing the measurable voluntary activation.
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