Senolytic clearance protocols are therapeutic interventions, often involving specific compounds or transient lifestyle modifications, designed to selectively induce apoptosis, or programmed cell death, in senescent cells—cells that have ceased dividing but remain metabolically active and secrete pro-inflammatory molecules. These protocols are a key strategy in longevity medicine, aiming to reduce the systemic “senescence-associated secretory phenotype” (SASP), which includes pro-inflammatory cytokines and altered hormonal signaling that drive chronic aging and disease. Efficient clearance is essential for tissue rejuvenation.
Origin
This field of research is a direct outgrowth of cellular aging and gerontology studies, which identified senescent cells as a major contributor to age-related tissue dysfunction and chronic inflammation. The term senolytic, meaning “destroying the aged,” describes the specific action of these agents. The protocols are rapidly evolving, with a focus on natural compounds and pharmaceuticals that transiently target the survival pathways of these cells.
Mechanism
The mechanism involves administering compounds that disrupt the anti-apoptotic pathways that senescent cells utilize to resist death, such as BCL-2 or PI3K/AKT. By selectively inducing apoptosis in these non-dividing, pro-inflammatory cells, the protocol reduces the overall burden of the SASP, which subsequently lowers systemic inflammation and restores more youthful hormonal signaling and tissue microenvironments, thereby supporting overall metabolic and endocrine function.
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