Senescence Rejection is a proactive biological and clinical strategy focused on preventing or reversing cellular senescence, a state where cells permanently stop dividing but remain metabolically active and secrete pro-inflammatory and tissue-degrading factors. This intervention targets the clearance of these senescent cells and the suppression of the Senescence-Associated Secretory Phenotype (SASP), thereby mitigating chronic inflammation and preserving tissue function. It is a core tenet of longevity medicine aimed at reducing biological aging.
Origin
This concept stems from the field of gerontology and molecular biology, where cellular senescence was identified as a key mechanism driving age-related decline and disease pathology. The term “Rejection” signifies an active therapeutic goal, utilizing senolytic agents or other modulators to purge these dysfunctional cells from the body. Hormonal balance is intrinsically linked, as endocrine decline often exacerbates the accumulation of senescent cells.
Mechanism
Senescence Rejection operates by activating apoptotic pathways specifically in senescent cells, leading to their programmed death and subsequent clearance by the immune system. Certain peptides and compounds can interfere with the anti-apoptotic mechanisms that senescent cells employ to survive, or they can directly suppress the detrimental SASP factors like inflammatory cytokines. By reducing the burden of these non-functional, pro-aging cells, tissue microenvironments are restored to a more regenerative state.
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