Senescence Hyposomatotropism Reversal is a clinical intervention strategy aimed at restoring the declining activity of the somatotropic axis, characterized by reduced pulsatile secretion of Growth Hormone (GH) and subsequent lower levels of Insulin-like Growth Factor 1 (IGF-1), which is a hallmark of biological aging. This reversal seeks to mitigate the adverse consequences of age-related GH deficiency, including sarcopenia, increased visceral adiposity, and reduced bone mineral density, thereby promoting an anabolic state and improving overall tissue vitality. The goal is to re-establish a youthful GH/IGF-1 signaling profile.
Origin
This term is rooted in geriatric endocrinology and longevity medicine, specifically addressing the phenomenon of ‘somatopause,’ the age-related decline in the growth hormone axis. The term ‘reversal’ signifies a therapeutic approach that actively counters this physiological decline, moving beyond mere replacement to a functional restoration of the axis’s output.
Mechanism
The reversal mechanism focuses on stimulating the hypothalamic-pituitary unit to increase the endogenous, pulsatile release of Growth Hormone, often through the use of Growth Hormone-Releasing Hormone (GHRH) analogues or Growth Hormone Secretagogues (GHSs). This enhanced pulsatility leads to increased GH secretion, which then stimulates the liver and other tissues to synthesize and secrete IGF-1. The resulting systemic increase in IGF-1 mediates the anabolic effects on muscle and bone, while also promoting lipolysis and modulating glucose metabolism, effectively countering the catabolic and degenerative changes associated with senescence.
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