Neural Network Redundancy is the physiological characteristic of the central nervous system where multiple, overlapping pathways and interconnected circuits exist to perform a single function, ensuring resilience and compensatory capacity in the face of injury, stress, or age-related decline. This is a measure of neurological robustness and functional reserve. High redundancy contributes directly to the maintenance of cognitive function despite underlying structural changes.
Origin
This concept is a fundamental principle of computational neuroscience and systems biology, recognizing that distributed processing is key to the brain’s remarkable reliability and adaptability. ‘Redundancy’ is viewed clinically as a protective factor against acute or chronic cognitive impairment, a concept often referred to as cognitive reserve. This understanding has shifted focus toward maintaining network connectivity rather than just individual cell health.
Mechanism
Redundancy is structurally supported by high synaptic density, a complex network of interneurons, and the ability of non-primary neural pathways to be recruited and strengthened through neuroplasticity mechanisms. Hormones, such as estrogen and progesterone, contribute by promoting the overall health and connectivity of these networks, enhancing the brain’s ability to reroute signals when a primary path is compromised. A highly redundant network translates to greater cognitive reserve and functional longevity.
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