Mental Procrastination, viewed endocrinologically, is a behavioral manifestation of impaired executive function and reduced dopaminergic signaling, often exacerbated by chronic stress and hormonal imbalance. It is the voluntary delay of necessary tasks, driven by a limbic system preference for immediate reward or avoidance of perceived threat. This pattern can be chemically reinforced by low-grade HPA axis activation and a resulting state of low cognitive energy.
Origin
While procrastination is a well-established psychological term, its application in this context links the behavioral pattern directly to underlying neurochemical and hormonal substrates. The connection is rooted in neuroendocrinology, recognizing that optimal cognitive drive and motivation are dependent on balanced levels of cortisol, dopamine, and thyroid hormones. It highlights the physiological basis of a common cognitive struggle.
Mechanism
The mechanism involves a functional disconnect between the prefrontal cortex, responsible for planning and goal-directed behavior, and the amygdala, which mediates fear and avoidance. High baseline cortisol can deplete dopamine stores and impair prefrontal cortex function, reducing the capacity for effortful task initiation. Low thyroid hormone levels also contribute by decreasing overall neuronal excitability and metabolic energy available for complex cognitive processing.
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