The biological process encompassing the growth, ossification, and structural remodeling of the mandible and associated facial skeletal structures, largely influenced by the pubertal surge and sustained presence of androgens, primarily testosterone. This development results in the characteristic sexually dimorphic features of the adult male face, including a broader, more angular jawline, a prominent chin, and increased bone density. It is a classic example of hormonal influence on skeletal morphology.
Origin
The term combines “male” (referring to sex), “jaw” (the mandible and maxilla), and “development” (the process of growth and differentiation). This concept is rooted in developmental biology, human anatomy, and endocrinology, specifically focusing on the effects of sex steroids on skeletal maturation. The process is a key element of secondary sexual characteristics.
Mechanism
The pubertal increase in testosterone acts directly on androgen receptors present in osteoblasts and osteoclasts within the mandibular bone tissue. This hormonal signaling promotes osteoblast activity, leading to periosteal bone apposition and increased bone mineral density, resulting in the characteristic squaring and forward projection of the male jaw. Furthermore, androgens influence the growth of associated musculature, such as the masseter, which also contributes to the overall robust appearance of the male lower face.
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