Low-Grade Inflammation Markers are measurable biological indicators, such as high-sensitivity C-Reactive Protein (hs-CRP), interleukin-6 (IL-6), or fibrinogen, whose subtle, chronically elevated levels signal a persistent, non-acute systemic immune activation. This low-grade, or smoldering, inflammation is scientifically recognized as a key driver of biological aging and is mechanistically linked to the development of insulin resistance, endothelial dysfunction, and the degradation of crucial hormonal receptor sensitivity. Clinical practice focuses intently on reducing these markers to mitigate chronic disease risk and enhance overall metabolic health.
Origin
This concept emerged from modern clinical pathology and immunometabolism research, which established a strong, quantifiable correlation between persistently elevated inflammatory cytokines and the pathogenesis of nearly all age-related chronic diseases. It provides a precise, measurable link between subtle, long-term immune system dysregulation and a compromised healthspan.
Mechanism
The chronic, low-level release of pro-inflammatory cytokines is often triggered by factors like excessive visceral adiposity, intestinal barrier dysfunction (gut dysbiosis), and chronic psychological stress, which collectively activate immune cells. These circulating cytokines then actively interfere with normal hormonal signaling, notably by impairing insulin receptor function and promoting tissue resistance to cortisol. Monitoring these markers allows for the objective clinical assessment of the body’s total inflammatory burden and the precise efficacy of targeted anti-inflammatory interventions.
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