A state where the volume and complexity of incoming data overwhelm the central nervous system’s capacity for effective processing and integration, leading to cognitive fatigue and heightened physiological stress reactivity. This saturation places an undue metabolic burden on neuronal resources, often manifesting as decreased mental bandwidth and impaired decision-making related to self-care behaviors. It represents a form of chronic, low-grade neurological overload.
Origin
This term originates from systems overload theory, where the capacity of a processing unit is exceeded by the input rate, applied to the human brain’s interaction with modern information environments. In the context of hormonal health, this overload is directly implicated in the sustained elevation of allostatic load markers. The saturation point signifies functional impairment of higher cognitive control.
Mechanism
Information saturation forces the brain into a high-arousal state, increasing sympathetic outflow and potentially driving chronic cortisol secretion due to the perceived inability to manage the input flow. This sustained activation depletes neurotransmitter precursors and exhausts the neural resources needed for tasks like accurate internal state monitoring. Consequently, metabolic flexibility and HPA axis responsiveness become compromised.
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