The specific neuro-regenerative and protective effects mediated by the copper-binding peptide Glycyl-L-Histidyl-L-Lysine (GHK-Cu) within the central and peripheral nervous systems. This peptide is recognized for its ability to promote the healing of damaged neural tissue, stimulate nerve growth factor (NGF) production, and exhibit potent anti-inflammatory and antioxidant properties in the brain. GHK-Cu Neural Repair represents a targeted strategy to restore neurological integrity following injury or age-related decline.
Origin
The peptide GHK was originally discovered in human plasma, and its copper-complexed form (GHK-Cu) was later found to possess remarkable tissue remodeling and wound-healing capabilities across various organ systems, including the skin. Its application to ‘Neural Repair’ stems from subsequent research demonstrating its ability to modulate genes involved in neuroprotection and regeneration, bridging regenerative medicine with neuroscience.
Mechanism
GHK-Cu acts by modulating the expression of numerous genes involved in tissue repair and anti-aging, effectively resetting the cellular environment. It promotes the breakdown of abnormal collagen and scar tissue, while simultaneously stimulating the synthesis of healthy tissue components and nerve growth factors. Its copper-binding capacity contributes to its potent antioxidant activity, protecting neuronal membranes from oxidative stress and supporting the structural scaffolding necessary for axon regeneration and synaptic remodeling.
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