Epigenetic Influence on Decline refers to the non-genetic mechanisms that alter gene expression and contribute significantly to the functional deterioration associated with biological aging. These influences include DNA methylation patterns, histone modifications, and non-coding RNA activity, which do not change the underlying DNA sequence but dictate which genes are transcriptionally active or silent. This concept acknowledges that environmental and lifestyle factors actively modulate the rate of age-related decline by progressively altering the epigenome.
Origin
This term is rooted in the field of epigenetics, which has fundamentally expanded the understanding of heredity and gene regulation beyond the classic Mendelian genetics model. The connection to “Decline” reflects modern longevity science’s focus on the epigenome as a key, measurable, and modifiable driver of healthspan and lifespan. Epigenetic clocks are now used clinically to estimate biological age based on the cumulative changes in these specific patterns.
Mechanism
The mechanism involves the progressive accumulation of aberrant epigenetic marks, such as global DNA hypomethylation and site-specific hypermethylation, leading to the dysregulation of critical gene expression profiles. This results in the silencing of genes essential for cellular repair and maintenance, while inadvertently activating genes that promote inflammation and senescence. Interventions in this area aim to restore a more youthful and stable epigenetic pattern, thereby decelerating the functional decline.
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