DHEA Adrenopause is a clinical term describing the progressive, age-related decline in the production of dehydroepiandrosterone (DHEA) and its sulfated form (DHEA-S) by the adrenal cortex, separate from gonadal senescence. DHEA is a critical precursor to sex hormones like testosterone and estrogen, and its reduction is a notable biological hallmark of aging that begins around the third decade of life. This specific hormonal shift is associated with a decrease in metabolic resilience, mood disturbances, and reduced bone mineral density. Recognizing this distinct pattern allows for targeted, personalized endocrine support strategies.
Origin
The term is a compound of DHEA, the adrenal steroid, and “adrenopause,” paralleling the term “menopause” but specifically referencing the adrenal gland’s diminished output. Its origin lies in the field of gerontology and endocrinology, where researchers identified a clear and predictable drop in circulating DHEA-S levels with advancing age. Early studies characterized DHEA as a potential biomarker of biological aging due to its significant concentration decrease over the adult lifespan. The concept helps differentiate age-related adrenal changes from primary adrenal insufficiency.
Mechanism
The mechanism involves a gradual reduction in the activity of key adrenal enzymes, particularly those responsible for the final steps of DHEA synthesis within the zona reticularis of the adrenal cortex. This decline is believed to be centrally regulated, potentially involving changes in the adrenal gland’s sensitivity to ACTH stimulation or age-related alterations in the upstream hypothalamic-pituitary signaling. As DHEA-S is the most abundant circulating steroid, its reduction impacts numerous physiological processes by limiting the substrate available for peripheral tissue conversion into active androgens and estrogens. Restoring DHEA levels aims to mitigate these downstream hormonal deficits.
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