This term defines the integrated set of immediate and delayed physiological adjustments the human body undertakes to maintain core temperature homeostasis when exposed to environmental cold stress. Key responses include peripheral vasoconstriction, shivering thermogenesis, and the non-shivering thermogenesis mediated by brown adipose tissue activation. Monitoring this response offers insight into metabolic health and autonomic regulation capacity. We observe significant endocrine shifts during this process.
Origin
The description is purely physiological, stemming from the need to counteract hypothermia. ‘Cold Exposure’ denotes the external stimulus, while ‘Physiological Response’ captures the body’s systemic reaction. This area of study is central to thermoregulation research in environmental physiology.
Mechanism
Initial exposure triggers sympathetic nervous system activation, leading to peripheral vasoconstriction to shunt warm blood centrally. If cold persists, the release of norepinephrine and thyroid hormones increases metabolic rate to generate heat. This process requires the coordinated action of the hypothalamus, adrenal glands, and sympathetic nerve endings to balance heat production against heat loss effectively.
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