Cognitive Friction Signals represent the subtle, measurable indicators of increased mental effort and reduced processing efficiency in the brain. These signals manifest as a delay in reaction time, a greater number of errors, or subjective feelings of mental strain during complex tasks. They are critical markers of compromised neurological reserve and can often be linked to underlying hormonal imbalances or metabolic stress.
Origin
This is a conceptual term emerging from the convergence of cognitive psychology, human-computer interaction, and applied neuroscience. The term ‘friction’ is a metaphor borrowed from physics, describing resistance to movement, which in this context refers to resistance in information flow within neural circuits. Clinically, it provides a functional endpoint for assessing cognitive load.
Mechanism
The generation of cognitive friction signals is mechanistically tied to the efficiency of neurotransmitter systems, particularly dopamine and acetylcholine, and the energetic status of prefrontal cortex neurons. When resources like ATP or key neurotransmitter precursors are depleted, the neural circuits required for sustained attention and executive function must expend greater energy to maintain performance. This heightened metabolic demand registers as the measurable friction.
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