Cellular Senescence Negation is a biological and clinical objective focused on inhibiting or reversing the state of irreversible growth arrest that cells enter, known as senescence. Senescent cells accumulate with age and hormonal decline, secreting pro-inflammatory molecules that damage surrounding tissue and accelerate aging—a phenomenon termed the Senescence-Associated Secretory Phenotype (SASP). Negating this process is a key strategy in longevity medicine to preserve tissue function and reduce chronic inflammation.
Origin
This term is a product of modern gerontology and molecular biology research, specifically building upon the discovery of telomere shortening and the role of p53/p21 pathways in inducing senescence. The concept of “negation” refers to the therapeutic strategies developed, such as senolytics (agents that kill senescent cells) and senomorphics (agents that suppress the SASP), which have emerged from this fundamental scientific understanding.
Mechanism
The negation mechanism involves targeting the specific survival pathways of senescent cells or neutralizing the detrimental effects of the SASP. Senolytic agents work by inducing apoptosis (programmed cell death) selectively in senescent cells, thereby clearing them from the tissue and reducing the overall inflammatory burden. This clearance promotes tissue renewal and restores the microenvironment, allowing healthy cells to proliferate and function optimally, directly supporting hormonal tissue vitality.
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