Clinically designed intervention programs focused on improving the function and longevity of individual cells, the foundational units of human physiology. These protocols specifically target the core processes of cellular aging, including mitochondrial dysfunction, telomere shortening, and DNA damage accumulation. The objective is to enhance cellular resilience, metabolic efficiency, and the capacity for self-repair, thereby slowing systemic aging.
Origin
This concept is deeply rooted in molecular biology and the emerging field of cellular senescence, translating fundamental scientific discoveries into clinical practice. It reflects the understanding that the health of the entire organism is a direct reflection of the health of its constituent cells. The term provides a clear framework for interventions that operate at the subcellular level.
Mechanism
The primary mechanism involves supporting mitochondrial integrity and biogenesis, ensuring efficient ATP production and minimizing oxidative stress. Protocols often utilize agents that activate autophagy, the cellular self-cleaning process, to remove damaged organelles and protein aggregates. Additionally, these strategies aim to maintain telomere length and enhance the activity of DNA repair enzymes, protecting the genomic stability that is vital for long-term cellular function.
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