This term refers to the intricate, measurable biological processes and hormonal shifts that underpin the subjective experience of anxiety. It involves the activation of the body’s defensive systems, specifically the hypothalamic-pituitary-adrenal or HPA axis, which is the central coordinator of the stress response. Understanding this physiology allows clinicians to distinguish between normal stress reactivity and a dysregulated state requiring targeted intervention. Chronic anxiety can lead to allostatic load, reflecting cumulative wear and tear on the body’s hormonal and metabolic systems.
Origin
The concept is derived from the integration of psychopathology, the study of the nervous system (neuroscience), and the study of the endocrine system (endocrinology). The understanding of the body’s internal reaction to perceived threat has evolved significantly since Walter Cannon’s description of the fight-or-flight response. Physiology itself stems from the Greek physis meaning “nature, origin” and logia meaning “study of.”
Mechanism
The mechanism initiates in the brain when a perceived threat stimulates the hypothalamus to release corticotropin-releasing hormone, or CRH. CRH subsequently prompts the pituitary gland to secrete adrenocorticotropic hormone, which travels via the bloodstream to the adrenal glands. This hormonal cascade culminates in the release of glucocorticoids, primarily cortisol, which mobilize energy resources and suppress non-essential functions like digestion and immune response. A negative feedback loop is normally engaged to restore hormonal equilibrium once the perceived threat subsides.
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