Age-Related Inflammatory Burden defines the cumulative physiological stress resulting from chronic, low-grade systemic inflammation that typically increases with chronological aging, a phenomenon known as “inflammaging.” This persistent inflammatory state is characterized by elevated circulating levels of pro-inflammatory markers like C-reactive protein and various cytokines. It represents a foundational driver of cellular senescence and the progressive decline of organ function, directly impacting hormonal health and longevity.
Origin
The concept originates from gerontology and immunology, specifically the observation that a sterile, low-grade inflammatory state is a universal feature of aging, even in the absence of acute infection. This systemic burden is considered a critical link between the aging process and the development of chronic diseases, including cardiovascular and neurodegenerative disorders. Clinically, quantifying this burden offers a measurable metric for assessing biological age and health trajectory.
Mechanism
The underlying mechanism involves the accumulation of senescent cells that secrete a senescence-associated secretory phenotype (SASP), alongside the gradual exhaustion and dysfunction of immune regulatory cells. These factors perpetuate a cycle of inflammation that disrupts cellular signaling, damages DNA, and promotes insulin resistance. This chronic disruption directly impairs endocrine glands, contributing to reduced hormone production and peripheral hormone receptor insensitivity, thereby accelerating the decline in hormonal vitality.
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